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Extrarenal azotemia in gastro-intestinal hemorrhage

(I) General and clinical consideration

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Summary

We have presented a summary of the work which has been done on the subject of extrarenal azotemia. This state may be produced by either an increased nitrogen catabolism or deficient nitrogen excretion.

It is fairly well proved that a state of extrarenal azotemia exists in many patients in whom there is acute gastro-intestinal hemorrhage. We have presented the essential facts in such a case and have reviewed the similar experience of other authors.

The maximum rise in the blood urea in these patients apparently takes place between the second and the fourth day after the onset of the hemorrhage with a gradual return to normal thereafter.

We have summarized the many theories as to the cause of this urea rise, and conclude that the number of theories offer proof of our lack of knowledge.

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Bibliography

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    Borst, J. G. G.: Cause of Hyperchloremia and Hyperazotemia in Patients with Recurrent Massive Hemorrhage from Peptic Ulcer.Acta. Med. Scandinav., 97:68–88, Oct., 1938.

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Correspondence to D. H. Kaump or J. C. Parsons.

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Kaump, D.H., Parsons, J.C. Extrarenal azotemia in gastro-intestinal hemorrhage. Jour. D. D. 7, 189–190 (1940). https://doi.org/10.1007/BF02997819

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Keywords

  • Peptic Ulcer
  • Uremia
  • Sudden Loss
  • Maximum Rise
  • Tarry Stool