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The role of the circulation in the production of peptic ulcer

Conclusion

From histologic study of 161 cases of acute focal lesions of the gastric mucosa, we would conclude.

  1. 1.

    Regardless of etiology, focal gastric lesions are the result of chronic circulatory insufficiency to all the structures of the gastric wall.

  2. 2.

    Such circulatory deficiency is part of a generalized insufficiency intensified by intrinsic vascular peculiarities of the stomach, of an anatomical or vasomotor nature.

  3. 3.

    Generalized circulatory insufficiency may be established through quantitative, qualitative or vasomotor alterations in the circulatory system.

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References

  1. 1.

    Reeves, T. B.: Collected Papers of The Mayo Clinic, 10: 3, 1918.

  2. 2.

    Weech, A. A. and Paige, B. H.:Am. J. Path., 13:249–256, March, 1937.

  3. 3.

    Nedzel, A. J.:Arch. Path., 26:988–1008, Nov., 1938.

Additional References

  1. Ferguson, A. E.:Am. J. Anat., 42:403–435, 1928.

  2. Robertson, H. E.:J. A. M. A., 112:22, Jan., 1939.

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Author information

Correspondence to Russell S. Boles or Helena E. Riggs or John O. Griffiths.

Additional information

Read at the Annual 1939 Session of the American Gastro-Enterological Association at Atlantic City.

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Boles, R.S., Riggs, H.E. & Griffiths, J.O. The role of the circulation in the production of peptic ulcer. American Journal of Digestive Diseases 6, 632–636 (1939). https://doi.org/10.1007/BF02996336

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Keywords

  • Peptic Ulcer
  • Gastric Mucosa
  • Gastric Lesion
  • Chronic Traumatic Encephalopathy
  • Chronic Ulcer